Papilloma virus on lips

Human warmth and odors pheromones are easily detected at that distance. Gesture air-kissing is a successful, hygienic way to avoid microbiologic transfer. Although this ancient custom implies high status, germ transfer through touching shaking hands or hand kissing combined with a lack of hand hygiene is cited as a reason for the spread of antibiotic-resistant nosocomial infections such as methicillin-resistant Staphylococcus.

Consequently, after any kissing, it is advisable to disinfect contact areas with detergent, a practice common in hospitals.

Cultural kissing involves facial contact other than with the lips, such as the Inuit kunik described earlier. Indonesian and African tribes promote frontal cranial contact as a greeting. Whether lips are closed or open is irrelevant, and the bodily contact is short.

The osculum is the closed-lip peck on the cheek. It is often combined with gestural kissing, repeated on both sides of the face. Some Russian cultures repeat the gesture more than once when greeting and taking farewells. The basium kiss is the mutual approximation of lips without opening the mouth. Slight pressure is momentarily maintained. Prolonging a basium kiss is reportedly satisfying to some.

Orogenital contact is considered a saviolum kiss and is a form of sexual expression that commonly occurs in all types of partnerships: heterosexual, homosexual, and lesbian.

Fellatio and cunnilingus are orogenital activities for sexual gratification. Viral transmission is facilitated through coitus, but some viruses, such as hpv , are also transmitted vertically from mother to child or through physical social activities such as hugging and play among siblings. Recurrent respiratory papillomatosis rrp , a hyperproliferative condition of the respiratory mucosal tree, is caused by hpv s 2. Kissing allows hpv s to infect other head-and-neck areas such as the oral and sinus cavities, the conjunctiva of the eyes, the ear canals, the oropharynx, and the tonsils.

Although those sites are more rarely infected than is the genital tract, the resulting rrp infections are challenging to comprehend, to examine, to thwart, and to stop. De novo anogenital warts in children should raise suspicions of sexual abuse by infected adults 2 , 3. These mucotropic viruses infect the head-and-neck mucosae, the upper aerodigestive tract, and the anogenital region. Some hpv s are deemed low-risk lr —that is, non-oncogenic for example, hpv 6 and hpv Their high-risk hr counterparts such as hpv 16, hpv 18, hpv 51, and hpv 53 have high oncogenic potential.

Each species consists of several different subspecies: lr hpv 11 has 10 subspecies; hr hpv 16 has 9; hr hpv 18 has 7; hr hpv 51 has 5; and hr hpv 53 has 6. Oncogenic types such as hr hpv 16 and hpv 18 cause high-grade dysplasias and neoplasias. The lr hpv s hpv 6 and 11 are far less prevalent in genital cancers, but might be associated with rrp infection 2 , 3.

All hpv s share common structural features that allow for the immune response to successful target most of them 3 , 4. Transmission of disease through kissing is accomplished by viral, bacterial, and rarely protozoal organisms 5.

Viral infections such as hepatitis, verruca vulgaris, Epstein—Barr viral mononucleosis 6 , and hiv are transmitted not only through close sexual contact, but also through exchange of bodily fluids saliva from kissing. Once considered rarely transmissible by saliva, hiv is now acknowledged to be transmissible during intimate kissing.

Neisseria meningitides, the organism that causes meningitis in the young, can be transmitted in the same manner. The hpv s—harboured by men and women alike—cause most but not all labial, vulval, perineal, penile, and oral papillomata, and dna typing shows that oral and cervical cancers derive from distinct, transmissible hpv s 7.

Preventing hpv disease is more desirable and effective than reversing established cellular reactions after hpv infection. Although there are natural constraints on hpv oncogenesis [such as production of the p53 and pRB retinoblastoma proteins], hr hpv E6 and E7 proteins can destabilize epithelial chromosomes.

Inactivation of those two major suppressors p53 and pRB and other cellular proteins largely accounts for oncogenic induction by the hr hpv s 3. A quadrivalent vaccine against genome-derived proteins—including the L1 virus-like particles from hpv s 6, 11, 16, and 18—constitutes effective immune prophylaxis against the development of hpv- associated cancers and mucosal warts.

Vaccines impart immunity against hpv- induced neoplastic change in most recipients 4 , 8. Because people practicing various forms of saviolum kissing transmit viable microbiota such as hpv , hiv , and so on , it is important to further examine the implications of infection by hr hpv s. Chronic infection with hr hpv is a necessary prerequisite for most cervical cancers, but invasive neoplasia does not always develop after infection with hr hpv. Although hr hpv s remain dormant for decades, cancer is uncommon among women and men because infection with hr hpv is frequently self-limiting.

A decade or more passes before neo-plastic changes transition to stage 1 dysplastic lesions carcinoma in situ or intraepithelial carcinoma, also called cin lesions.

Approximately 7 percent of Americans ages 14 to 69 have oral HPV. The number of people who have oral HPV has increased over the past three decades.

HPV is considered a high-risk type. Oropharyngeal cancer is rare. Approximately 1 percent of people have HPV Less than 15, people get HPV-positive oropharyngeal cancers each year. Age is a risk factor for oropharyngeal cancer. No test is available to determine if you have HPV of the mouth. Your dentist or doctor may discover lesions through a cancer screening, or you may notice the lesions first and make an appointment. If you have lesions, your doctor can perform a biopsy to see if the lesions are cancerous.

If HPV is present, the cancer may be more responsive to treatment. Most types of oral HPV go away before they cause any health issues.

If you develop oral warts due to HPV, your doctor will likely remove the warts. Treating the warts with topical treatments can be difficult because the warts may be hard to reach. Your doctor may use any of the following methods to treat the warts:. If you do develop oropharyngeal cancer, treatment options are available.

HPV-positive oropharyngeal cancers have better outcomes and fewer relapses after treatment than HPV-negative cancers. Treatment for oropharyngeal cancer can include radiation therapy, surgery, chemotherapy, or a combination of these.

Lifestyle changes are some of the easiest ways to help prevent HPV. Here are some tips for prevention:. People aged 15 and over get three shots over six months. This vaccine was previously only available to people up until age The most common areas in the head and neck that develop HPV cancers are the tonsil and the base of tongue. In fact, HPV is estimated to cause 70 percent of the cancers that arise in these areas.

Symptoms of cancers in this region include pain, intermittent bleeding out of the mouth, ear pain, a foreign body sensation in the throat, and if the cancer spreads to the neck lymph nodes, a new lump that persists in the neck.

If you suspect there is something wrong, make an appointment with an ear, nose, and throat doctor, otherwise known as a otolaryngologist. The otolaryngologist will perform a physical examination to search for suspicious lesions.